| ID | Type | Location (GRCh37) | Location (GRCh38) | Length | GC content |
|---|---|---|---|---|---|
| hsa_FNDC3B_0008100 | Intron-Exon-Intron | chr3:171830242-171979873 | chr3:172112452-172262083 | 250 nt | 0.3600 |
| hsa_FNDC3B_0008900 | Exon-Exon | chr3:171830242-172003774 | chr3:172112452-172285984 | 153 nt | 0.4641 |
| hsa_FNDC3B_0009000 | Exon-Exon | chr3:171830242-172013304 | chr3:172112452-172295514 | 152 nt | 0.4737 |
| hsa_FNDC3B_0009100 | Exon-Exon | chr3:171830242-172016577 | chr3:172112452-172298787 | 352 nt | 0.4631 |
| hsa_FNDC3B_0009200 | Exon-Exon | chr3:171830242-172028671 | chr3:172112452-172310881 | 392 nt | 0.4898 |
| hsa_FNDC3B_0009300 | Exon-Exon | chr3:171830246-171851336 | chr3:172112456-172133546 | 211 nt | 0.4787 |
| hsa_FNDC3B_0009500 | Exon-Exon | chr3:171830246-171944737 | chr3:172112456-172226947 | 77 nt | 0.4935 |
| hsa_FNDC3B_0009400 | Exon-Exon | chr3:171830246-172028671 | chr3:172112456-172310881 | 1278 nt | 0.4859 |
| hsa_FNDC3B_0009600 | Exon-Exon | chr3:171830336-172013254 | chr3:172112546-172295464 | 206 nt | 0.4951 |
| hsa_FNDC3B_0009700 | Intron-Intron | chr3:171842815-171843543 | chr3:172125025-172125753 | 729 nt | 0.4746 |
A study in mice demonstrated that the circFNDC3B was significantly downregulated in hearts post-myocardial infarction and in human cardiac tissues of ischemic cardiomyopathy patients, where its overexpression in cardiac endothelial cells increased vascular endothelial growth factor expression, enhanced angiogenic activity, and reduced apoptosis [Tian et al. DOI:10.3390/molecules26041155].