| ID | Type | Location (GRCh37) | Location (GRCh38) | Length | GC content |
|---|---|---|---|---|---|
| hsa_FNDC3B_0012200 | Intron-Intron | chr3:171916760-171917709 | chr3:172198970-172199919 | 950 nt | 0.4232 |
| hsa_FNDC3B_0012300 | Intron-Intron | chr3:171921112-171923430 | chr3:172203322-172205640 | 2319 nt | 0.4036 |
| hsa_FNDC3B_0012400 | Intron-Exon-Intron | chr3:171923269-172028671 | chr3:172205479-172310881 | 54 nt | 0.4259 |
| hsa_FNDC3B_0012500 | Intron-Intron | chr3:171931226-171931443 | chr3:172213436-172213653 | 218 nt | 0.3532 |
| hsa_FNDC3B_0013300 | Intron-Exon-Intron | chr3:171944661-171969482 | chr3:172226871-172251692 | 754 nt | 0.4695 |
| hsa_FNDC3B_0013100 | Intron-Exon-Intron | chr3:171944661-171979873 | chr3:172226871-172262083 | 250 nt | 0.3440 |
| hsa_FNDC3B_0013500 | Exon-Exon | chr3:171944661-172003774 | chr3:172226871-172285984 | 136 nt | 0.4559 |
| hsa_FNDC3B_0013600 | Exon-Exon | chr3:171944661-172013304 | chr3:172226871-172295514 | 77 nt | 0.4935 |
| hsa_FNDC3B_0013400 | Exon-Exon | chr3:171944661-172025291 | chr3:172226871-172307501 | 1013 nt | 0.4906 |
| hsa_FNDC3B_0013700 | Exon-Exon | chr3:171944661-172048505 | chr3:172226871-172330715 | 175 nt | 0.4971 |
A study in mice demonstrated that the circFNDC3B was significantly downregulated in hearts post-myocardial infarction and in human cardiac tissues of ischemic cardiomyopathy patients, where its overexpression in cardiac endothelial cells increased vascular endothelial growth factor expression, enhanced angiogenic activity, and reduced apoptosis [Tian et al. DOI:10.3390/molecules26041155].