| ID | Type | Location (GRCh37) | Location (GRCh38) | Length | GC content |
|---|---|---|---|---|---|
| hsa_FNDC3B_0017300 | Exon-Exon | chr3:171969050-172046866 | chr3:172251260-172329076 | 871 nt | 0.4811 |
| hsa_FNDC3B_0017400 | Exon-Exon | chr3:171969050-172052872 | chr3:172251260-172335082 | 1272 nt | 0.4772 |
| hsa_FNDC3B_0017500 | Exon-Exon | chr3:171969050-172064230 | chr3:172251260-172346440 | 1856 nt | 0.4914 |
| hsa_FNDC3B_0017600 | Exon-Exon | chr3:171969050-172070873 | chr3:172251260-172353083 | 2287 nt | 0.4945 |
| hsa_FNDC3B_0017700 | Exon-Exon | chr3:171969050-172098883 | chr3:172251260-172381093 | 2795 nt | 0.4891 |
| hsa_FNDC3B_0019800 | Exon-Exon | chr3:172003716-172016577 | chr3:172285926-172298787 | 271 nt | 0.4354 |
| hsa_FNDC3B_0019900 | Exon-Exon | chr3:172003716-172028671 | chr3:172285926-172310881 | 464 nt | 0.4655 |
| hsa_FNDC3B_0020800 | Exon-Exon | chr3:172013153-172028671 | chr3:172295363-172310881 | 405 nt | 0.4741 |
| hsa_FNDC3B_0021600 | Exon-Exon | chr3:172016518-172028671 | chr3:172298728-172310881 | 253 nt | 0.4743 |
| hsa_FNDC3B_0021700 | Exon-Exon | chr3:172016518-172052872 | chr3:172298728-172335082 | 779 nt | 0.4763 |
A study in mice demonstrated that the circFNDC3B was significantly downregulated in hearts post-myocardial infarction and in human cardiac tissues of ischemic cardiomyopathy patients, where its overexpression in cardiac endothelial cells increased vascular endothelial growth factor expression, enhanced angiogenic activity, and reduced apoptosis [Tian et al. DOI:10.3390/molecules26041155].