Basic Information
Symbol
circMACF1
RNA Class
circRNA
Alias
Microtubule Actin Crosslinking Factor 1
KIAA0754
ABP620
ACF7
Actin Cross-Linking Family Protein 7
Trabeculin-Alpha
KIAA0465
KIAA1251
Lnc-PMIF
MACF
Postulated Migration Inhibitory Factor
620 KDa Actin Binding Protein
FLJ45612
FLJ46776
Microtubule-Actin Cross-Linking Factor 1, Isoforms 1/2/3/4/5
Microtubule-Actin Cross-Linking Factor 1, Isoforms 6/7
Microtubule-Actin Cross-Linking Factor 1
Uncharacterized Protein KIAA0754
620 KDa Actin-Binding Protein
Actin Cross-Linking Factor
Macrophin 1
Macrophin-1
LIS9
OFC4
Location (GRCh37)
1:39547089-39951524
UCSC Genome Browser
Location (GRCh38)
1:39081417-39485852
Host gene
MACF1
Host category
mRNA
Forensic tag(s)
Sudden cardiac death diagnosis
External database
Transcripts
| ID | Type | Location (GRCh37) | Location (GRCh38) | Length | GC content |
|---|---|---|---|---|---|
| hsa_MACF1_0018400 | Exon-Exon | chr1:39757582-39784287 | chr1:39291910-39318615 | 292 nt | 0.5308 |
| hsa_MACF1_0018500 | Exon-Exon | chr1:39757582-39788680 | chr1:39291910-39323008 | 99 nt | 0.5051 |
| hsa_MACF1_0018600 | Exon-Exon | chr1:39757582-39793025 | chr1:39291910-39327353 | 378 nt | 0.4206 |
| hsa_MACF1_0018700 | Exon-Exon | chr1:39757582-39812882 | chr1:39291910-39347210 | 129 nt | 0.5271 |
| hsa_MACF1_0018800 | Exon-Exon | chr1:39757582-39838268 | chr1:39291910-39372596 | 207 nt | 0.4831 |
| hsa_MACF1_0018900 | Exon-Exon | chr1:39757582-39854330 | chr1:39291910-39388658 | 129 nt | 0.5271 |
| hsa_MACF1_0019100 | Exon-Exon | chr1:39758495-39760822 | chr1:39292823-39295150 | 288 nt | 0.4375 |
| hsa_MACF1_0019200 | Intron-Intron | chr1:39758516-39758802 | chr1:39292844-39293130 | 287 nt | 0.3624 |
| hsa_MACF1_0019400 | Exon-Exon | chr1:39759130-39760822 | chr1:39293458-39295150 | 267 nt | 0.4494 |
| hsa_MACF1_0019500 | Exon-Exon | chr1:39759130-39927709 | chr1:39293458-39462037 | 155 nt | 0.4516 |
Showing 321 to 330 of 794 entries
Forensic Context
In mice, circMACF1 was found to be downregulated in myocardial tissue following myocardial infarction, and its experimental overexpression was shown to restore cardiomyocyte apoptosis and reduce myocardial infarct size [Tian et al. DOI:10.3390/molecules26041155].