A study in humans with coronary artery disease identified the miR-6862-5p as downregulated in whole blood of patients with recurrent myocardial infarction after percutaneous coronary intervention, with this differential expression remaining significant after false-discovery rate correction in a replication cohort, demonstrating its potential as a biomarker for thrombotic risk [Onuoha et al. DOI:10.1111/cts.13307].